EMF Blocking and EMF Shielding
No matter if you live in a house or apartment or simply need to ensure that your house is free of electromagnetic fields, there are a number options to limit your exposure. One of the easiest is to reduce your use of electronic devices. https://www.pearltrees.com/maracadrum8/item512699682 is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another easy way to protect your house from EMF radiation is to put up a shielding canopy for RF. This is a fabric made of net that has EMF shielding. It's used to block EMFs from entering a space. Another option is to have your home equipped with a conductive enclosure. These devices are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF produces antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation of cancer stem cells. This could explain the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on cancer patients is not clear.
The effects of AM RF EMF on HCC tumor growth in vivo was studied in mice. Discover more were split into 3 groups. One group did not have exposure to RF EMF. Find more information were exposed to RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumours treated with HCCMF were significantly shrinking. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism of tumour-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is controlled by CACNA1H the protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H may have broader implications for treatment and diagnosis of different cancers.
The tumours in those in the group that were unaffected RF EMF, and were fed a normal diet of mice. The tumors in the HCCMF group were treated with Huh7 cells at the time they were five-seven weeks old. The tumors were then killed when they showed excessive burden.
The tumors of the three groups showed distinct growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after 8 weeks. However, the tumors that were treated using RCF showed no shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis that is common when tumors are exposed to RCF. There is a possibility that the necrosis was caused by a lack of oxygen in the more invasive tumors.
In conclusion, the findings indicate that AM RF EMF exhibits anticancer properties in vitro as well as in the vivo. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors for HCC patients. It is possible that AM RF EMF produces these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting effect on the development of HCC tumours in vivo.